Hypokalemia

For every 0.1 unit increase in arterial pH, serum potassium levels can drop by approximately 0.5 mEq/L due to this shift alone. (Na/K ATPase causes K to go into the cells and H+ to move out of the cells to maintain normal blood pH)

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If a patient receives 10mg of furosemide daily, it leads to around 0.5 mmol/L fall in serum potassium.

At standard therapeutic does of adrenaline infusion, initially serum K rises (due to release from live) an then serum potassium falls by 0.7 mmol/L daily due to intracellular shift of K (activation of Na/K ATPase). 3 Sodium move out of cell and 2 K move into the cells, and so Na rises by 2mmol/L daily with standard therapeutic treatment.

Amikacin causes a 0.4 mEq drop in potassium levels over a 7 day treatment course.

Vancomycin causes a 1 mEq drop over 3 days treatment 

Aminophylline causes a transcellular shift of 0.5 mEq at standard treatment doses  over 1 hour. And it reverses over next 2 to 3 hours.

Caffeine citrate, a methylxanthine like aminophylline, can cause hypokalemia through a combination of a transcellular shift (moving potassium into cells) and renal loss (excreting potassium in urine). 0.5 mmol/L fall over 2 hours, that reverses over next 3 to 6 hours.

Salbutamol nebulization at standard doses or albuterol infusion at standard doses causes 0.5 mmol/L over 2 hours, and it reverts over 3 to 4 hours.

An infusion of 10 units of R insulin causes fall of 1 mmol/L over 1 hour by transcellular shifts, and it reverts over next 3 to 4 hours.